Affecting   1 billion people worldwide, hypertension remains the most common, readily identifiable, and reversible risk factor for myocardial infarction, stroke, heart failure, atrial fibrillation, aortic dissection, and peripheral arterial disease. Because of escalating obesity and population aging, the global burden of hypertension is rising and projected to affect 1.5 billion persons”one third of the world™s population”by the year 2025.High BP remains the leading cause of death worldwide and one of the world™s great public health problems.

Hypertension currently is defined as a usual BP of 140/90 mm Hg or higher.

Essential hypertension is the term applied to the 95% of hypertensive patients in which elevated blood pressure results from complex interactions between multiple genetic and environmental factors. The proportion regarded as essential will diminish with improved detection of clearly defined secondary  causes and with better understanding of pathophysiology.

Exacerbating factors include obesity, sleep apnea, increased salt intake, excessive alcohol use,cigarette smoking, polycythemia, nonsteroidal anti-inflammatory (NSAID) therapy, and low potassium intake.

Obesity is associated with an increase in intravascular volume, elevated cardiac output, activation of the renin-angiotensin system and, probably, increased sympathetic outflow. Weight reduction lowers blood pressure modestly.(so you have to reduce the weight of pt by sedating yin or yang or unit humidity as per type and pt should be advised to do enough walk )

Increased salt intake probably increases blood pressure in some individuals so dietary salt restriction is recommended in patients with hypertension .(Advise patients to reduce their salt intake to less than 4 g of salt a day (approximately 1550 mg of sodium a day), which is approximately one teaspoon of salt).

Excessive use of alcohol also raises blood pressure, perhaps by increasing plasma catecholamines. Hypertension can be difficult to control in patients who consume more than 40 g of ethanol (two drinks) daily or drink in binges.(So patient should stop alcohol to less than 60 ml per day or use sujok for deaddiction)

Cigarette smoking raises blood pressure. .(So patient should stop cigarette smoking or use sujok for deaddiction).

Aerobic exercise lowers blood pressure in previously sedentary individuals, but increasingly strenuous exercise in already actives subjects has less effect.(so advice Pt to do mild exercises only. Brisk walking will reduce BP but running may increase BP.)

Reduce stress and or any mind imbalance by using Triorigin or Six Ki treatment.

If lipid profile of Pt is high then reduce lipids by six ki ——-E(circulatory system)-E(hemic system)-IV sedate or E(circulatory system)-E(hemic system)-D(lipids)-II,III sedate and V,VI tone.

Low potassium intake is associated with higher blood pressure in some patients; an intake of 90 mmol/d is recommended so if kidney function test I.e creatinine level is normal then pt should take coconut water daily.

If patient has diabetes or Insulin resistance then it is better to treat them first and later treat hypertension as treating insulin resistance may simply reduce the BP.

Secondary Hypertension is one which has  Identifiable causes of hypertension like

  1. Sleep apnea
  2. Drug-induced or drug-related
  3. Chronic kidney disease
  4. Primary aldosteronism
  5. Renovascular disease
  6. Long-term corticosteroid therapy and Cushing syndrome
  7. Pheochromocytoma
  8. Coarctation of the aorta
  9. Thyroid or parathyroid disease

You should rule out the above mentioned causes of hypertension. These will be dealt in part 2 of this article.


Different types of essential hypertension and pathomechanics behind them is-

  1. SYSTOLIC HYPERTENSION IN YOUNG ADULTS- At one end of the age spectrum is isolated systolic hypertension (ISH) in young adults (typically 17 to 25 years of age). The key hemodynamic abnormalities are increased cardiac output and a stiff aorta, both presumably reflecting an overactive sympathetic nervous system.




How to reach Sympathetic NS?

III rent II (NS rent NS),II rent Ah III (PNS),Ah III rent Urn III (Vegetative NS),Um III rent Ah III (Sympathetic NS)-sedate III,tone VI


F(vessels of circulatory system)-sedate AH III(sympathectonia of vessels)

If above formula not working then E-I,III sedation should work as cardiac output is high and pulse rate is high.

2.DIASTOLIC HYPERTENSION IN MIDDLE AGE. When hypertension is diagnosed in middle age (typically, 30 to 50 years of age), the most common BP pattern is elevated diastolic pressure, with systolic pressure being normal (isolated diastolic hypertension) or elevated (combined systolic-diastolic hypertension). This pattern constitutes classic essential hypertension. Isolated diastolic hypertension is more common in men and is often associated with middle-age weight gain. Without treatment, isolated diastolic hypertension often progresses to combined systolic-diastolic hypertension. The fundamental hemodynamic fault is an elevated systemic vascular resistance coupled with an inappropriately normal cardiac output. Vasoconstriction at the level of the resistance arterioles results from increased neurohormonal drive and an autoregulatory reaction of vascular smooth muscle to an expanded plasma volume, the latter because of impairment in the kidneys™ ability to excrete sodium.

F(vessels of circulatory system)-sedate AH III(sympathectonia of vessels)

H-II tone,V sedate

H-I(nephrons of kidney)-II tone,V sedate

Functional order tonification of kidneys in triorigin

Very good stimulation of heart,brain and kidney(by kidney beans) correspondence points.


3.ISOLATED SYSTOLIC HYPERTENSION IN OLDER ADULTS-After the age of 55 years, ISH (systolic BP >140 mm Hg and diastolic BP <90 mm Hg) is the most common form. In developed countries, systolic pressure rises steadily with age; in contrast, diastolic pressurerises until about 55 years of age, then falls progressively thereafter. The resultant widening of pulse pressure indicates stiffening of the central aorta and a more rapid return of reflected pulse waves from the periphery, causing an augmentation of systolic aortic pressure . Accumulation of collagen (which is poorly distensible)adversely affects its ratio to elastin in the aortic wall.

HO is body, ho is layer ,no is communication system, he is artery “ ho sedation in arterites.



Baroreceptors and Hypertension-watch the video link from the URL below or visit medisujok on face book for deep knowledge on baroreceptors.

Baroreceptors together with cardiovascular control centre in medulla of brain stem are very important in regulating the BP. (So once a SJT knows the position of baroreceptors in chest(on aortic arch) and neck (bilaterally on carotid sinus) then he should give very good correspondence application for these areas. Also pressing medulla -the lower portion of brainstem-with intention to hit cardiovascular control centre will give a dramatic result.)





baroreceptor 2



Renal Mechanisms– In many forms of experimental and human hypertension, the fundamental abnormality is an acquired or inherited defect in the kidney™s ability to excrete the excessive sodium load imposed by a modern diet high in salt. As humans evolved in a low-sodium/high-potassium environment, the human kidney is ill-equipped to handle the current exposure to high sodium and low potassium. Renal sodium retention expands the plasma volume, increasing cardiac output and triggering autoregulatory responses that increase systemic vascular resistance. Salt retention also augments the smooth muscle contraction produced by endogenous vasoconstrictors. Beyond raising BP, a high-salt diet also accelerates hypertensive target organ damage.. Hypertensive individuals excrete the same amount of a given dietary sodium load as normotensive individuals do, but at a higher BP, and require many more hours to excrete the sodium load and to achieve sodium balance.

H-II tone,V sedate

H-I(nephrons of kidney)-II tone,V sedate

Functional order tonification of kidneys in triorigin

ENDOTHELIAL CELL DYSFUNCTION- The endothelial lining of blood vessels is critical to vascular health and constitutes a majordefense against hypertension. Dysfunctional endothelium is characterized by impaired release of endothelium-derived relaxing factors (e.g., nitric oxide, endothelium-derived hyperpolarizing factor) and enhanced release of endothelium-derived constricting, proinflammatory, prothrombotic, and growth factors.Arterial layers

HO is body,He is system,HO of HO is circulatory system where Ho is structure of vessels in which NE is Intima in which Ne is endothelium which is secreting less relaxing factors  (He e.g., nitric oxide) and more constricting factors(Ho) so –tone HE, sedate HO, tone NE in endothelium of blood vessels.

VASCULAR REMODELING– Vasoconstriction initiates small artery remodeling, which normalizes wall stress. Normal smooth muscle cells rearrange themselves around a smaller lumen diameter, a process termed inward eutrophic remodeling

REMODELLING of blood vessels

HO is body, ho is layer ,no is communication system, he is artery “ ho sedation in arterites.


Hormonal Mechanisms: Renin-Angiotensin-Aldosterone System – Activation of the renin-angiotensin-aldosterone system (RAAS) is one of the most important mechanisms contributing to endothelial cell dysfunction, vascular remodeling, and hypertension.


RAS system of kidney.


AS per Dr Violettas version-to stop renin release H(kidney)-I(nephron)-E(renal corpuscle)-III(renin) sedate.

Also press correspondence of adrenals to reduce release of aldosterone from it.

LATEST research on hypertension– Due to investigations into the pathophysiology of hypertension, both in animals and humans, growing evidence suggests that hypertension may have an immunological basis. Studies have revealed that hypertension is associated with renal infiltration of immune cells and that pharmacologic immunosuppression (such as with the drug mycophenolate mofetil) or pathologic immunosuppression (such as occurs with HIV) results in reduced blood pressure in animals and humans. Evidence suggests that T lymphocytes and T-cell derived cytokines (eg, interleukin 17, tumor necrosis factor alpha) play an important role in hypertension.

So reach immune system sedate He, tone Ho, tone Ne or do anti functional sedation of immune system joint,also do same in thymus and adrenals. Dont forget to press correspondence of Thymus and Adrenals.                                                or

On reaching the circulatory system (also kidney as internal organ) when we rent hetero functional system opens in which Ne is immune system in which sedate He tone Ho tone Ne.


The emergence of new invasive procedures for lowering of sympathetic activity and BP in patients with refractory hypertension”catheter-based radiofrequency ablation of the renal sympathetic nerves after reaching the renal artery ablate”has rekindled excitement about neural mechanisms, even before the procedures have approval by the Food and Drug Administration.


renal sympathetic denervation


Reach kidney open He as function system in which He is nervous system of which He is sympathetic nervous system and ho is parasympathetic system so sedate sympathetic nervous system of kidney.

Or H-intention needle on J-E(renal artery)- intention needle on J-I(sympathetic nerves)-sedate I,II,III and tone IV,V,VI.


                                               SOME TIME TESTED FORMULAS

Yang ajna hotness sedate or J III sedate and K(G)VI tone if BP more then 170/105 .

If Bp less then 170/105 yin ajna hotness sedate .

In young people high stable BP E “III sedate but if fluctuating BP then F “III sedate.

If obese person or high lipid profile  sedateIII, IV,V in E or F.

In older people sedate V,VI in E usually.

Balance the constitution-if done perfectly will bring BP down.

Dont forget essence of sujok i.e Smile Taichi and SMILE MEDITATION which is a must in patients treatment and his upgradation.
ONLY if patient understands that this is just a role given to him by Creator and his role can be upgraded on understanding the concept of Universal Drama he can be cured.